repli g ultra fast mini kit (Qiagen)
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repli g ultra fast mini kit
Repli G Ultra Fast Mini Kit, supplied by Qiagen, used in various techniques. Bioz Stars score: 94/100, based on 161 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/repli+g+ultra+fast+mini+kit/REPLI-g+UltraFast+Mini+Kit/pmc07387320-58-16-21
Average 94 stars, based on 161 article reviews
Repli G Ultra Fast Mini Kit, supplied by Qiagen, used in various techniques. Bioz Stars score: 94/100, based on 161 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/repli+g+ultra+fast+mini+kit/REPLI-g+UltraFast+Mini+Kit/pmc07387320-58-16-21
Average 94 stars, based on 161 article reviews
repli g ultra fast mini kit - by Bioz Stars,
2026-10
94/100 stars
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Biomarker Discovery:Article Title: T-cell acute lymphoblastic leukemia in infants has distinct genetic and epigenetic features compared to childhood cases. Article Snippet: T-Cell Acute Lymphoblastic Leukemia in Infants Has Distinct Genetic and Epigenetic Features Compared to Childhood Cases Mareike Doerrenberg 1 , Andreas Kloetgen 1,2 , Kebria Hezaveh 1 , Wilhelm Wössmann 3 , Kirsten Bleckmann 4 , Martin Stanulla 5 , Martin Schrappe 6 , Alice C McHardy 2 , Arndt Borkhardt 1 and Jessica I Hoell 1,* 1 Department of Pediatric Oncology, Hematology and Clinical Immunology, Heinrich-Heine- University, Medical Faculty, Düsseldorf, Germany 2Computational Biology of Infection Research, Helmholtz Center for Infection Research, Braunschweig, Germany 3Department of Pediatric Hematology and Oncology, University Hospital Gießen and Marburg, Gießen, Germany 4 ALL BFM Trial center, University Hospital Schleswig-Holstein, Kiel, Germany 5Pediatric Hematology and Oncology, Hannover Medical School, Hannover, Germany 6 Department of Pediatrics, University Medical Center Schleswig-Holstein, Kiel Campus, Kiel, Germany * Correspondence to: Jessica Hoell, Department of Pediatric Oncology, Hematology and Clinical Immunology, Heinrich-Heine- University Düsseldorf, Moorenstrasse 5, 40225 Düsseldorf, Germany; phone: +49 211 81 17680, fax: +49 211 81 16707, e-mail: jessica.hoell@med.uni-duesseldorf.de Running title: Genetic and epigenetic features in infant T-ALL This article has been accepted for publication and undergone full peer review but has not been through the copyediting, typesetting, pagination and proofreading process which may lead to differences between this version and the Version of Record.. Please cite this article as an ‘Accepted Article’, doi: 10.1002/gcc.22423 This article is protected by copyright.. All rights reserved. Amplification:Article Title: T-cell acute lymphoblastic leukemia in infants has distinct genetic and epigenetic features compared to childhood cases. Article Snippet: T-Cell Acute Lymphoblastic Leukemia in Infants Has Distinct Genetic and Epigenetic Features Compared to Childhood Cases Mareike Doerrenberg 1 , Andreas Kloetgen 1,2 , Kebria Hezaveh 1 , Wilhelm Wössmann 3 , Kirsten Bleckmann 4 , Martin Stanulla 5 , Martin Schrappe 6 , Alice C McHardy 2 , Arndt Borkhardt 1 and Jessica I Hoell 1,* 1 Department of Pediatric Oncology, Hematology and Clinical Immunology, Heinrich-Heine- University, Medical Faculty, Düsseldorf, Germany 2Computational Biology of Infection Research, Helmholtz Center for Infection Research, Braunschweig, Germany 3Department of Pediatric Hematology and Oncology, University Hospital Gießen and Marburg, Gießen, Germany 4 ALL BFM Trial center, University Hospital Schleswig-Holstein, Kiel, Germany 5Pediatric Hematology and Oncology, Hannover Medical School, Hannover, Germany 6 Department of Pediatrics, University Medical Center Schleswig-Holstein, Kiel Campus, Kiel, Germany * Correspondence to: Jessica Hoell, Department of Pediatric Oncology, Hematology and Clinical Immunology, Heinrich-Heine- University Düsseldorf, Moorenstrasse 5, 40225 Düsseldorf, Germany; phone: +49 211 81 17680, fax: +49 211 81 16707, e-mail: jessica.hoell@med.uni-duesseldorf.de Running title: Genetic and epigenetic features in infant T-ALL This article has been accepted for publication and undergone full peer review but has not been through the copyediting, typesetting, pagination and proofreading process which may lead to differences between this version and the Version of Record.. Please cite this article as an ‘Accepted Article’, doi: 10.1002/gcc.22423 This article is protected by copyright.. All rights reserved. Article Title: 5-Azacytidine modulates CpG methylation levels of EZH2 and NOTCH1 in myelodysplastic syndromes. Article Snippet: Purpose Molecular mechanisms of response to hypomethylating agents in patients with myelodysplastic syndromes (MDS) and chronic myelomonocytic leukemia (CMML) still remain largely unknown.. Therefore, the effects of 5-Azacytidine (Aza) on clonal architecture and DNA methylation were investigated in this study.. Methods Using next-generation sequencing (NGS), 30 myeloid leukemia-associated genes were analyzed in 15 MDS/ CMML patients with excellent response to Aza. Article Title: EZH2 mutations and promoter hypermethylation in childhood acute lymphoblastic leukemia. Article Snippet: functional consequence of EZH2 promoter hypermethylation on H3K27me3 was studied by Western blot analyses of primary cells.. Results Loss-of-function EZH2 mutations were detected in 2/152 (1.3 %) patients with common-ALL and early T-cell precursor (ETP)-ALL, respectively.. In one patient, targeted deep sequencing identified cooperating mutations in ASXL1 and TET2. Article Title: Sequencing and comparison of the Rickettsia genomes from the whitefly Bemisia tabaci Middle East Asia Minor I. Article Snippet: The whitefly, Bemisia tabaci, harbors the primary symbiont ‘Candidatus Portiera aleyrodidarum’ and a variety of secondary symbionts.. Among these secondary symbionts, Rickettsia is the only one that can be detected both inside and outside the bacteriomes.. Infection with Rickettsia has been reported to influence several aspects of the whitefly biology, such as fitness, sex ratio, virus transmission and resistance to pesticides. Whole Genome Amplification:Article Title: Frequent ASXL1 mutations in children and young adults with chronic myeloid leukemia. Article Snippet: In the last decade a large number of somatic mutations affecting multiple pathways have been identified in myeloid malignancies with varying frequencies and combinations that overlap the different disease entities [1].. Initially, aberrations were discovered in genes that confer a growth advantage by altering signaling pathways and expression of key transcriptional targets.. Over time, additional pathways such as epigenetic modification, RNA splicing, and the cohesin complex were found to be involved in myeloid leukemogenesis. Article Title: 5-Azacytidine modulates CpG methylation levels of EZH2 and NOTCH1 in myelodysplastic syndromes. Article Snippet: Purpose Molecular mechanisms of response to hypomethylating agents in patients with myelodysplastic syndromes (MDS) and chronic myelomonocytic leukemia (CMML) still remain largely unknown.. Therefore, the effects of 5-Azacytidine (Aza) on clonal architecture and DNA methylation were investigated in this study.. Methods Using next-generation sequencing (NGS), 30 myeloid leukemia-associated genes were analyzed in 15 MDS/ CMML patients with excellent response to Aza. Article Title: EZH2 mutations and promoter hypermethylation in childhood acute lymphoblastic leukemia. Article Snippet: functional consequence of EZH2 promoter hypermethylation on H3K27me3 was studied by Western blot analyses of primary cells.. Results Loss-of-function EZH2 mutations were detected in 2/152 (1.3 %) patients with common-ALL and early T-cell precursor (ETP)-ALL, respectively.. In one patient, targeted deep sequencing identified cooperating mutations in ASXL1 and TET2. Article Title: CYP2C76 deficiency is embryonic lethal in cynomolgus macaques: The potential role of CYP2C76 in early embryogenesis. Article Snippet: Cynomolgus macaques are an important primate species for drug metabolism studies, however cynomolgus CYP2C76, an important drug-metabolizing enzyme, accounts for drug metabolism differences to humans, so CYP2C76-null animals might show drug-metabolizing properties more similar to humans.. In this study, attempts were made to produce CYP2C76-null animals by assisted reproduction technology.. Oocytes and sperm collected from the heterozygotes for the null allele (c.449TG > A) were subjected to intracytoplasmic sperm injection, and the embryos produced were cultured in vitro through the blastocyst stage. Article Title: Prevalence and dynamics of clonal hematopoiesis caused by leukemia-associated mutations in elderly individuals without hematologic disorders Article Snippet: Samples of 50 healthy elderly individuals were genotyped for a panel of 30 commonly mutated leukemia-associated genes by targeted deep NGS [ , ], using the 454 GS Junior platform (Roche Diagnostics, Mannheim, Germany) at a sensitivity level of 5%. .. Prior to sequencing, whole genome amplification (WGA) was performed using 20 ng template gDNA and the Next-Generation Sequencing:Article Title: 5-Azacytidine modulates CpG methylation levels of EZH2 and NOTCH1 in myelodysplastic syndromes. Article Snippet: Purpose Molecular mechanisms of response to hypomethylating agents in patients with myelodysplastic syndromes (MDS) and chronic myelomonocytic leukemia (CMML) still remain largely unknown.. Therefore, the effects of 5-Azacytidine (Aza) on clonal architecture and DNA methylation were investigated in this study.. Methods Using next-generation sequencing (NGS), 30 myeloid leukemia-associated genes were analyzed in 15 MDS/ CMML patients with excellent response to Aza. DNA Amplification:Article Title: EZH2 mutations and promoter hypermethylation in childhood acute lymphoblastic leukemia. Article Snippet: functional consequence of EZH2 promoter hypermethylation on H3K27me3 was studied by Western blot analyses of primary cells.. Results Loss-of-function EZH2 mutations were detected in 2/152 (1.3 %) patients with common-ALL and early T-cell precursor (ETP)-ALL, respectively.. In one patient, targeted deep sequencing identified cooperating mutations in ASXL1 and TET2. Sequencing:Article Title: Prevalence and dynamics of clonal hematopoiesis caused by leukemia-associated mutations in elderly individuals without hematologic disorders Article Snippet: Samples of 50 healthy elderly individuals were genotyped for a panel of 30 commonly mutated leukemia-associated genes by targeted deep NGS [ , ], using the 454 GS Junior platform (Roche Diagnostics, Mannheim, Germany) at a sensitivity level of 5%. .. Prior to sequencing, whole genome amplification (WGA) was performed using 20 ng template gDNA and the Multiple Displacement Amplification:Article Title: Sequencing and comparison of the Rickettsia genomes from the whitefly Bemisia tabaci Middle East Asia Minor I. Article Snippet: The whitefly, Bemisia tabaci, harbors the primary symbiont ‘Candidatus Portiera aleyrodidarum’ and a variety of secondary symbionts.. Among these secondary symbionts, Rickettsia is the only one that can be detected both inside and outside the bacteriomes.. Infection with Rickettsia has been reported to influence several aspects of the whitefly biology, such as fitness, sex ratio, virus transmission and resistance to pesticides. |